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Prenatal alcohol exposure is associated with a range of adverse offspring neurodevelopmental outcomes. Several studies suggest that PAE modifies DNA methylation in offspring cells and tissues, providing evidence for a potential mechanistic link to Fetal Alcohol Spectrum Disorder.
Individuals with Fetal Alcohol Spectrum Disorder (FASD) are at risk of having adverse childhood experiences (ACEs), especially those with child protection or justice system involvement. The complex relationship between FASD and psychosocial vulnerabilities in the affected individual is an important clinical risk factor for comorbidity.
This audit aimed to increase understanding of the long-term outcomes of evidence-based medical and surgical interventions to improve gross motor function in children and adolescents with Cerebral Palsy.
Jonathan Raewyn Carol Carapetis AM Mutch Bower AM MBBS FRACP FAFPHM PhD FAHMS MBChB., DipRACOG., Cert.HPRT, FRACP., PhD MBBS MSc PhD FAFPHM DLSHTM
Barriers in addressing FASD in Australia include a drinking culture and large populations living in regional or remote communities with high risk populations.
This study examines the association between PAE in the general antenatal population and child neurodevelopment at 2 years, accounting for relevant factors.
To examine the association between dose, frequency, and timing of prenatal alcohol exposure and craniofacial phenotype in 12-month-old children.
Children of mothers with alcohol use disorders are at risk of not meeting minimum educational benchmarks in numeracy and literacy, with the risk highest among Indigenous children.
About a third of young people in youth detention in Western Australia have Fetal Alcohol Spectrum Disorder (FASD), data has found.
A higher than expected proportion of children with fetal alcohol spectrum disorders had gross motor scores that indicated impairment and need for therapy